Complex Odontomas Hampering Eruption of Permanent Tooth

Iran J Pediatr; Vol 24 (No 4), Aug 2014 Published by: Tehran University of Medical Sciences (http://ijp.tums.ac.ir) Fig.1: Left. NeuN-immunonegative neurons in a pontine histological section of a SIDS victim dead at 2 months. Right. NeuN_immunopositivity in an equivalent section of a control case (late fetal death -38 gestational weeks) for comparison NeuN immunostaining. Magnification: 40× chemical breathing-depended components of blood and cerebrospinal fluid (e.g. pO2, pCO2, pH). The pathological involvement of the vagalglossopharingeal neuronal circuitry explains some lethal reflexogenic mechanism, quoted in the literature as “drive”, “death-feigning” and/or “Ondina’s course” reflexes. Moreover, parahypoglossal glossopharingeal brainstem damage can promote suffocation by tongue hypotonia during supine sleep, especially if an abnormal reticular formation 4 impairs respiration and/or gastro-esophageal reflux goes into trachea. Overall the results obtained in the 12 cases of sudden death reported here, in agreement with those highlighted in our previous study[2] performed on a very large survey of perinatal deaths (140 SIDS, 95 SIUDS and 78 controls), show the common congenital origin of SIUDS and SIDS and support the statement of the National Institute of Child Health and Human Development: “SIDS is a developmental disorder. It takes its origin from fetal development”. In the world, only Italy has a national law that orders “Regulations for Diagnostic Post Mortem Investigation in Victims of the SIDS and Unexpected Fetal Death”. This law can be an example for the other nations in the world, and we believe it should be known. The present paper is the first result of the application of the law. The province of Trento (North Italy) applies this law because it is an autonomous province. The use of NeuN immunohistochemistry is another new important marker in the study of this pathology.